Clinical Acupuncture for Applied Kinesiologists Module 2 Part 3

Course overview

Explain how NF-kB activation drives the neuroinflammatory cascade and why inhibiting it is superior to NSAID prostaglandin blockade. Describe the five endogenous pathogenic factors and the eight principal patterns used in TCM inflammation diagnosis. Differentiate the pathogenic characteristics of wind, cold, and dampness and their effects on qi and body regions. Describe the triple burner's three divisions and its role in regulating fluid metabolism and Yuan Qi distribution. Identify the four short-term inflammation diagnostic protocols and the role of I6 factor insalivation in confirming findings. Demonstrate the clinical application of active range of motion, tactile recognition, flexor withdrawal, and nociceptive challenge testing. Apply supine gait testing of the periaqueductal gray and dorsal root ganglion to assess long-term neuroinflammation. Explain the cholinergic anti-inflammatory reflex and demonstrate sternal notch testing with latissimus dorsi in supine gait. Integrate the four short-term and three long-term inflammation protocols into a sequential two-minute examination. Apply metabolic, acupuncture, chiropractic, and low-level laser treatments to address short-term inflammation findings. Evaluate research supporting acupuncture's anti-inflammatory and neuroprotective effects in stroke and traumatic brain injury. Describe how meridian tracing and low-frequency electrostimulation influence BDNF and post-stroke motor recovery. Demonstrate meridian flushing and navel therapy localization to identify excess yin or yang energy by side. Recognize the clinical clues of meridian reversal and identify causes of negative pulse diagnosis findings. Identify the B&E acupoints on the head and their corresponding neurotransmitter associations. Apply Dr. Deal's five-finger acupuncture screening and integrate mental-emotional overlays into meridian treatment.