UNDERSEA & HYPERBARIC MEDICINE JOURNAL VOLUME 42, ISSUE #4

Course overview

LEARNER OBJECTIVES: As a result of having attended the meeting, the participants should have had the opportunity to: 1. Explain the role of ambient air in diving when surface air supplied system is used. 2. Discuss the gas kinetics under both normobaric conditions and increased pressure. 3. Understand Carbon monoxide (CO) poisoning and the effect in neurological and neuropsychological sequelae, including visual problems. SUBJECT MATTER: 1.CO and CO2 analysis in the diving gas of the fishermen of the Yucatan Peninsula. 2.Delayed visual disturbances in carbon monoxide poisoning: Identification and evaluation. TEACHING METHODS: ¿Journal Reading ¿Examination of material ¿Evaluation of activity EVALUATION METHODS: Evaluation is offered through the online educational portal with 3 graded questions each UHM Journal paper and an overall evaluation at completion of the Journal-based activity. REFERENCES: 1. Hampson NB, Weaver LK. Carbon monoxide poisoning:a new incidence for an old disease. Undersea Hyperb Med. 2007 May-Jun;34(3):163-168. 2. Weaver LK, Hopkins RO, Chan KJ, et al. Hyperbaric oxygen for acute carbon monoxide poisoning. N Engl J Med. 2002;347(14):1057-1067. 3. Weaver LK. Clinical practice. Carbon monoxide poisoning. N Engl J Med. 2009 Mar 19;360(12):1217-1225. 4. Simmons IG, Good PA. Carbon monoxide poisoning causes optic neuropathy. Eye (Lond). 1998;12 (Pt 5):809-814. 5. Amitai Y, Zlotogorski Z, Golan-Katzav V, Wexler A, Gross D. Neuropsychological impairment from acute low- level exposure to carbon monoxide. Arch Neurol. 1998 Jun;55(6):845-848. 6. Fine RD, Parker GD. Disturbance of central vision after carbon monoxide poisoning. Aust N Z J Ophthalmol. 1996 May;24(2):137-141. 7. Ersanli D, Yildiz S, Togrol E, Ay H, Qyrdedi T. Visual loss as a late complication of carbon monoxide poisoningand its successful treatment with hyperbaric oxygen therapy. Swiss Med Wkly. 2004 Oct 30;134(43-44):650-655. 8. Senol MG, Yildiz S, Ersanli D, et al. Carbon monoxide- induced cortical visual loss: treatment with hyperbaric oxygen four years later. Med Princ Pract. 2009;18(1):67-69. 9. Cockerham GC, Lemke S, Glynn-Milley C, Zumhagen L, Cockerham KP. Visual performance and the ocular surface in traumatic brain injury. Ocul Surf. 2013 Jan;11(1):25-34. 10. Wong AM. Eye movement disorders. Oxford University Press, USA; 2008. 11. Skenduli-Bala E, de Voogd S, Wolfs RC, et al. Causes of incident visual field loss in a general elderly population: the Rotterdam study. Arch Ophthalmol. 2005Feb;123(2):233-238. 12. Martinez-Thompson JM, Diehl NN, Holmes JM, Mohney BG. Incidence, types, and lifetime risk of adult- onset strabismus. Ophthalmology. 2014 Apr;121(4):877-882. 13. Cooper J, Jamal N. Convergence insufficiency-a major review. Optometry. 2012 Apr 30;83(4):137-158. 14. Lara F, Cacho P, Garc¿a A, Meg¿as R. General binocular disorders: prevalence in a clinic population. Ophthalmic Physiol Opt. 2001 Jan;21(1):70-74. 16. Kapoor N, Ciuffreda KJ. Vision disturbances following traumatic brain injury. Curr Treat Options Neurol. 2002 Jul;4(4):271-280. 17. Sarno S, Erasmus LP, Lippert G, Frey M, Lipp B, Schlaegel W. Electrophysiological correlates of visual impairments after traumatic brain injury. Vision Res. 2000;40(21):3029-3038 18. Suchoff IB, Kapoor N, Ciuffreda KJ, Rutner D, Han E, Craig S. The frequency of occurrence, types, and character- istics of visual field defects in acquired brain injury: a retro- spective analysis. Optometry. 2008 May;79(5):259-265. 19. Ramrattan RS, Wolfs RC, Panda-Jonas S, et al. Prevalence and causes of visual field loss in the elderly and associations with impairment in daily functioning: the Rotterdam Study. Arch Ophthalmol. 2001 Dec;119(12):1788-1794. (And more, limited by space)